Biology is designed for multi-semester biology courses for science majors. It is …
Biology is designed for multi-semester biology courses for science majors. It is grounded on an evolutionary basis and includes exciting features that highlight careers in the biological sciences and everyday applications of the concepts at hand. To meet the needs of today’s instructors and students, some content has been strategically condensed while maintaining the overall scope and coverage of traditional texts for this course. Instructors can customize the book, adapting it to the approach that works best in their classroom. Biology also includes an innovative art program that incorporates critical thinking and clicker questions to help students understand—and apply—key concepts.
By the end of this section, you will be able to:Describe physical …
By the end of this section, you will be able to:Describe physical and chemical immune barriersExplain immediate and induced innate immune responsesDiscuss natural killer cellsDescribe major histocompatibility class I moleculesSummarize how the proteins in a complement system function to destroy extracellular pathogens
This resource is a video abstract of a research paper created by …
This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview:
"Pancreatic ductal adenocarcinoma is one of the most lethal cancers, with a five-year survival rate of only 9%. Inflammation-mediated tissue damage plays a role in pancreatic cancer, and chronic pancreatitis is a risk factor for cancer development. Unfortunately, exactly how inflammation drives cancer initiation remains unclear. A recent study identified a novel signaling pathway connecting inflammation to pancreatic cancer. In a mouse model of chronic pancreatitis, repeatedly inducing pancreatic inflammation accelerated tumor development. Immune cells including M2 macrophages and eosinophils were recruited to fibrotic regions of the pancreas, and expression of the cytokine IL-5, which can recruit these inflammatory cells, increased in response to inflammation. Pancreatic cells upregulated the receptor for IL-5 upon transformation to tumor cells, which increased tumor cell motility..."
The rest of the transcript, along with a link to the research itself, is available on the resource itself.
No restrictions on your remixing, redistributing, or making derivative works. Give credit to the author, as required.
Your remixing, redistributing, or making derivatives works comes with some restrictions, including how it is shared.
Your redistributing comes with some restrictions. Do not remix or make derivative works.
Most restrictive license type. Prohibits most uses, sharing, and any changes.
Copyrighted materials, available under Fair Use and the TEACH Act for US-based educators, or other custom arrangements. Go to the resource provider to see their individual restrictions.
