Students will compare and contrast different perspectives of the French Revolution as depicted in two works of art. Students will discuss the use of satire and caricature to comment on historical and current events and will create satirical cartoons based on contemporary issues.

Students will analyze art elements and symbolism in a late-19th-century painting, create a self-portrait, and learn a dance depicted in the painting

Students will compare and contrast how two late-19th-century paintings depict celebrations in different ways through the artists' use of satire and color. Students will explore the historical context surrounding both canvases and create a painting of a celebration employing artistic techniques learned from the pictures.

A mild satire on Jackson and his Cabinet, portraying in imaginative terms a White House reception of popular French dancer and actress Madame Celeste. Seated in chairs in a White House parlor are six cabinet members. In the center Jackson sits behind a table, as "Door Keeper" Jimmy O'Neal (standing) presents Madame Celeste. The cabinet members are (left to right): Secretary of the Navy Mahlon Dickerson, Attorney General Benjamin F. Butler, Secretary of War Lewis Cass, Postmaster General Amos Kendall, Treasury Secretary Levi Woodbury, and Vice-President Martin Van Buren. Each figure's remarks are an amusing reflection of his own character or reputation. Dickerson: I never felt the inconvenience of being a bachelor untill now. what I have lost!! she as gracefull as a Seventy-four under full sail. Cass: This is a very strange introduction to the Cabinet when weighty matters are under discussion; but it does not become me to complain. Jimmy O'Neal: O' she'll bother them all by the powers faith, except my friend Kendal he has no soule for a pretty woman... Celeste: Mon General, if it is "glory enough" to serve under you "ma foi" vat is my grand satisfaction to see you wis de Grand Cabinet of dis Grand Nation here assamble. Jackson: Charming Creature. I've not lost all my penchant for pretty women .... Kendall: I wonder how the General could ever prefer the heels to the head. He never learnt that from me. But the least said the soonest mended. Woodbury: She has grace enough to dance all the surplus Revenue out of the Treasury Hasn't she, Mr. Attorney general? Butler: She is well enough, but I have conscientious scruples on these matters. Van Buren: Pooh pooh Butler, this is not the age for scruples of any kind. I like her rapid movements, her quick changes, her gracefull transitions. She is of my school ..." Weitenkampf's association of the cartoon with the Peggy Eaton affair of 1831, where several cabinet members resigned, is mistaken since the cabinet shown here consists of later appointees. The print appears from the style and monogram to be the work of lithographic draftsman Albert Hoffay.|Entered . . . 1836 by H.R. Robinson.|Published April 1836 by H.R. Robinson 48 Cortlandt St. N.Y.|Signed: A.H. (A.A. Hoffay?).|Title appears as it is written on the item.|Century, p. 40.|Weitenkampf, p. 40.|Forms part of: American cartoon print filing series (Library of Congress)|Published in: American political prints, 1766-1876 / Bernard F. Reilly. Boston : G.K. Hall, 1991, entry 1836-6.

This cell crossword puzzle uses vocabulary from CELLS alive! If you have trouble, use the "Search this Site" engine in the lefthand menu.

During development from stem to fully differentiated, cells in the body alternately divide (mitosis) and "appear" to be resting (interphase). This sequence of activities exhibited by cells is called the cell cycle. Watch this animation to learn more about each of the stages in the cell cycle: interphase, gap 0, gap 1, S Phase, gap 2, and M phase.

This cell structure crossword puzzle uses vocabulary from CELLS alive! If you have trouble and need a hint, use the "Search this Site" engine in the lefthand menu. Good Luck!

This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview:

"Apoptosis, a form of programmed cell death, plays critical roles in animal development and in repair of DNA damage. Since DNA damage is a major factor in cancer development, identifying the regulators of damage-induced apoptosis could help researchers develop treatments. A recent study investigated whether NHR-14, an important developmental protein in the model organism C. elegans, also contributes to damage-induced apoptosis . using mutant C. elegans that are especially susceptible to radiation-induced DNA damage. Deletion of the gene encoding NHR-14, which corresponds to HNF4 in humans, decreased radiation-induced apoptosis of sex cells without affecting the levels of normal (non-damage-induced) apoptosis, indicating a specific role in the damage-induced death pathway. Further exploration revealed that the NHR-14 gene acts “downstream” of the DNA damage checkpoint pathway and regulates the transcription of the genes egl-1 and ced-13 after DNA is damaged..."

The rest of the transcript, along with a link to the research itself, is available on the resource itself.

This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview:

"Critical defects that compromise the nucleus during cell division could be the basis for the age-accelerating effects of people living with progeria. Hutchinson-Gilford progeria syndrome is a genetic disorder that causes premature aging. Affecting one in 8 million newborns worldwide, the disorder is extremely rare—and fatal. The rapid aging of the cardiovascular system causes death due to heart attack or stroke in patients by their mid-teens. Progeria is caused by a tiny point mutation in the lamin A gene. This gene is responsible for producing structural proteins called lamins, which form the scaffolding that holds the cell nucleus together. The mutated form of prelamin A called progerin destabilizes the cell nucleus—the genetic control center of cells. The result is the fast-aging effects observed in progeria. But the link from gene mutation to physical disorder has remained a mystery. Previous studies have looked only at models of progeria, not at actual patient cells..."

The rest of the transcript, along with a link to the research itself, is available on the resource itself.

This illustration is an overview of cellular respiration connecting glycolysis to the Krebs (Citric Acid) Cycle and Oxidative Phosphorylation. The energy yield (ATP) and electron carriers (NADH and FADH2) are also shown in this overview.

This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview:

"G protein-coupled receptor (GPCR) signaling plays an important role in regulating cellular responses to external stimuli. GPCRs are so critical that they are a common pharmaceutical target, with 35% of available drugs targeting the receptors. But despite their importance, the mechanism underlying G protein selectivity for closely related Gαi proteins is unclear. In a recent study, researchers followed up on their previous finding that Gαi protein subunits prefer different lipid domains in the membrane. Using live-cell fluorescence microscopy techniques, they characterized the diffusion of Gαi subunits and the dopamine D2 long receptor isoform (D2R). They found that although Gαi protein subunits are very similar, the Gαi2 subunit displayed faster lateral diffusion than Gαi1. Distinct Gαi heterotrimers localized to different areas of the cell membrane, correlating with the efficiency of D2R-mediated inhibition of cAMP..."

The rest of the transcript, along with a link to the research itself, is available on the resource itself.

This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview:

"In many cell types, growth factor stimulation triggers the formation of structures called circular dorsal ruffles (CDRs). CDRs are large, rounded ruffles on the upper cell membrane that serve as platforms for PI3K–PIP3–AKT protein signaling and probably play a role in cell growth. CDRs are present in some types of cancer cells, but it’s unclear whether they contribute to cancer development. To find out, researchers recently treated six cancer cell lines and one normal cell line with two growth factors: epidermal growth factor and insulin. Both growth factors induced CDR formation in the Hep3B hepatocellular carcinoma line, but not in normal liver cells, other hepatocellular carcinoma cells, breast cancer cells, or pancreatic cancer cells. Closer analysis confirmed that growth factor receptor proteins were recruited to the CDRs and that the PI3K–PIP3–AKT pathway was activated at the ruffles..."

The rest of the transcript, along with a link to the research itself, is available on the resource itself.

Outlines the proposed structure and properties of the plasma membrane in mammalian cells and identifies cell compartments that are separated from each other by at least one plasma membrane.

Andy Meal – lecturer in Biological Sciences, School of Nursing, Midwifery and Physiotherapy, University of Nottingham

As taught Semesters 1 and 2, 2011

This learning object describes the structure and function of mammalian cell organelles. It is used as part of the level 1 Biological Sciences module delivered by the School of Nursing, Midwifery and Physiotherapy.

Dr Andy Meal, Lecturer in Biological Sciences, School of Nursing, Midwifery and Physiotherapy

This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview:

"The liver has a unique ability to regenerate after damage due to injury or disease. This regeneration relies primarily on the proliferation of hepatocytes, the main cells in the liver. Hepatocytes and many other liver cell types, including endothelial cells, bile duct cells, and immune cells, interact in a coordinated manner to enable repair. For example, liver endothelial cells promote the hepatocyte proliferation that’s necessary for regeneration, and hepatocytes activate a signaling pathway that promotes blood vessel growth to nourish the new liver tissue. If hepatocyte proliferation is impaired, an alternate pathway can direct liver regeneration through transformation of bile duct cells into hepatocytes. Liver endothelial cells promote this conversion process, helping to build the hepatocyte population. while connective tissue–forming cells encourage bile duct cell growth and proliferation, ensuring an ample supply of these cells..."

The rest of the transcript, along with a link to the research itself, is available on the resource itself.

This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview:

"Membrane-enclosed extracellular vesicles, exosomes, are a critical part of intercellular communication in many biological systems. However, the regulation and biological implications of exosome excretion and uptake remain unclear. A recent study examined the role of cellular retinoic acid (RA) binding protein (Crabp1) in exosome secretion and its relationship to receptor interacting protein 140 (RIP140), a pro-inflammatory transcription co-regulator. Crabp1 knockout mice consistently showed deficits in negative control of exosome secretion and exhibited increased vulnerability to systemic inflammation. Crabp1 knockout mice had significantly elevated RIP140-containing exosomes in their blood and cerebrospinal fluid. Cell culture experiments suggested that exosome secretion can transfer RIP140 from neurons to macrophages, where it promotes macrophage inflammatory polarization..."

The rest of the transcript, along with a link to the research itself, is available on the resource itself.

Student Atlas of Oregon in English and Spanish
Atlas of Oregon Climate and Climate Change

Poster showing St. George slaying the dragon; illustration forms a border around details of the patriotic rally. Title from item.

The Central Nervous System (CNS) is composed of the brain and the spinal cord. This page is specifically focussed on the histologic appearance, for anatomy see Forebrain, Midbrain, Hindbrain, Cranial Nerves, Spinal Cord and CNS Development.