Self-Management and Amygdala Triggers
(View Complete Item Description)Learn about amygdala triggers and how you can control them.
Material Type: Lesson
Learn about amygdala triggers and how you can control them.
Material Type: Lesson
A comprehensive review of the behavior or humans as well as other animals will be provided through each chapter. Topics such as learning, memory, emotions, behavioral disorders, and personality will be explored. Section I: Introduction to Psychology Section II: The Science of Psychology Section III: Physiological Aspects of Psychology Section IV: States of Consciousness Section V: Sensation and Perception Section VI: Growth and Development Section VII: Learning Section VIII: Thinking and Intelligence Section IX: Memory Section X: Emotion and Motivation Section XI: Personality Section XII: Social Psychology Section XIII: Psychological Disorders Section XIV: Treatment of Psychological Disorders
Material Type: Textbook
Unit Objectives - Enhancing Your Communication Skills *Explain how communications skills impact overall emotional intelligence *Explain various communication styles and identify your own “preferred” style *Describe the significance of nonverbal communication when communicating with others Unit Objectives - Ethical Behavior in the Workplace *Define ethics and give examples of ethical decisions you make in your daily life *Explain the levels of ethics and how they relate to human relations *Describe the models that an individual can use for ethical decision making.
Material Type: Homework/Assignment, Unit of Study
This course explores the social relevance of neuroscience, considering how emerging areas of brain research at once reflect and reshape social attitudes and agendas. Topics include brain imaging and popular media; neuroscience of empathy, trust, and moral reasoning; new fields of neuroeconomics and neuromarketing; ethical implications of neurotechnologies such as cognitive enhancement pharmaceuticals; neuroscience in the courtroom; and neuroscientific recasting of social problems such as addiction and violence. Guest lectures by neuroscientists, class discussion, and weekly readings in neuroscience, popular media, and science studies.
Material Type: Full Course
Introductory psychology course developed through the Ohio Department of Higher Education OER Innovation Grant. The course is part of the Ohio Transfer Module and is also named OSS015. For more information about credit transfer between Ohio colleges and universities please visit: www.ohiohighered.org/transfer. Team Lead Vincent Granito Lorain County Community College Content Contributors Nicole Brandt Columbus State Community College Lynne Gabriel Lakeland Community College Jackie Sample Central Ohio Technical College Librarian Rachel Dilley Columbus State Community College Review Team Melissa Beers Ohio State University Brian Gerber Stark State College
Material Type: Full Course
23 Things is a suite of 23 self-paced online modules that cover a range of topics from video editing to basic coding. Each module or 'thing' consists of information, interactive activities, and invitations to try out various open and free software applications and technologies. The modules have been created using H5P and can be downloaded individually as a single H5P file, modified and re-used under a CC-BY-SA licence - simply click on the 'reuse' link at the bottom of each module. The content was created by Curtin University students as part of a 'students as partners' project.
Material Type: Full Course, Interactive
Includes two interactive slide decks that provide overviews of some of the most useful collections of instructional resources, one for grades K-5, the other for grades 6-12. There are many more resource collections out there: these are just the "greatest hits"! Also includes collections of questions that can be used to engage students in Media Literacy.
Material Type: Lesson Plan, Teaching/Learning Strategy
Digital literacy is more than finding articles or being able to use Powerpoint - it's a flexible engagement within the digital world. This workshop uses poetry as an engagement lens on mindsets, learning, creativity and literacies. Developed as part of CAUL's Digital Dexterity launch program in 2019, this blackout poetry resource provides you with workshop slides, workshop plan and an instructional handout.
Material Type: Activity/Lab, Lesson Plan
The Living Book of Digital Skills (You never knew you needed until now) is a living, open source online guide to 'modern not-quite-technical computer skills' for researchers and the broader academic community.
Material Type: Textbook
An historical examination and analysis of the evolution and development of games and game mechanics. Topics include a large breadth of genres and types of games, including sports, game shows, games of chance, schoolyard games, board games, roleplaying games, and digital games. Students submit essays documenting research and analysis of a variety of traditional and eclectic games. Project teams required to design, develop, and thoroughly test their original games.
Material Type: Full Course
This textbook was written for a community college introductory course in spreadsheets utilizing Microsoft Excel. While the figures shown utilize Excel 2016, the textbook was written to be applicable to other versions of Excel as well. The book introduces new users to the basics of spreadsheets and is appropriate for students in any major who have not used Excel before.
Material Type: Textbook
Academic research articles have a structure and language that is different from our other reading materials such as textbooks. This lesson can help students new to academic research understand these differences and learn strategies for finding information in such articles.
Material Type: Activity/Lab, Assessment, Homework/Assignment, Module
This resource will provide college students with the skills necessary to find academic articles in support of a research writing assignment.This resource is written for a college level reader. The exercises are intended to be accomplished by using your college or university library website and the research resources on it. While this resource can be accomplished independently, it has been written to serve as instruction within a research methods course and assumes that students have an actual research project to which they can apply these strategies.
Material Type: Activity/Lab, Homework/Assignment, Reading, Student Guide
This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview: "Breast cancer is a leading cause of cancer-related death among women worldwide. One process that fuels the proliferation, survival, and migration of breast cancer cells is aerobic glycolysis. In normal cells, this conversion of glucose into lactate is normally reserved for low-oxygen conditions. Researchers have now identified a tumor-suppressing protein that helps regulate aerobic glycolysis in breast tumors. HRD1 is an enzyme known to suppress breast cancer proliferation and invasion. Experiments on human breast cancer cells clearly showed that upregulation of HRD1 decreased aerobic glycolysis. and subsequently inhibited breast cancer proliferation and invasion. This effect was found to be mediated by the enzyme PFKP. PFKP downregulation allowed HRD1 to inhibit the aerobic glycolysis and spread of breast cancer cells. These findings point to a new regulatory role for HRD1 and offer a possible target for future breast cancer therapies..." The rest of the transcript, along with a link to the research itself, is available on the resource itself.
Material Type: Diagram/Illustration, Reading
This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview: "RhoB is a rescue enzyme activated by toxins, stress, and inflammation and has been found to enhance the clearance of UTI-causing E. coli. Given these roles, researchers wondered whether RhoB might be a useful signal of other diseases—namely, ulcerative colitis. To find out, the team examined colon tissue from patients with ulcerative colitis, as well as mice with drug-induced colitis. They discovered that RhoB was enhanced in both humans and mice, with gut microbes contributing to that increase. Unlike in other studies, here RhoB was found to play an antagonistic role: compared with control mice (WT), colitis tended to recede in mice with low RhoB (RhoB-/-). A similar trend was observed in organs artificially grown from the tissue of mice without the gene that codes for RhoB. In these RhoB-less organoids, cells showed an increased ability to repair colon tissue compared with tissue from untreated control mice..." The rest of the transcript, along with a link to the research itself, is available on the resource itself.
Material Type: Diagram/Illustration, Reading
This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview: "Breast cancer is the most common type of cancer in women worldwide. More than 70% of breast cancers are estrogen receptor alpha (ERα) positive, indicating that they might be treatable with endocrine therapy. However, half of patients with ERα-positive breast cancer develop endocrine resistance, a major roadblock to successful therapy. In a new study, researchers sought to learn more about ERα-positive breast cancer to support the development of better treatments. The enzyme TRIM3, a regulator of protein stability vs. breakdown, was upregulated in ERα-positive breast cancer tissues, and high TRIM3 expression was associated with poor survival in patients receiving endocrine therapy. In experiments on mice and cell lines, TRIM3 promoted ERα signaling and was required for cancer growth and migration. Specifically, the filamin/NHL domain of TRIM3 bound to the DNA-binding domain of ERα in the nucleus..." The rest of the transcript, along with a link to the research itself, is available on the resource itself.
Material Type: Diagram/Illustration, Reading
This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview: "Colorectal cancer is the most common cancer worldwide and among the top 3 causes of cancer-related death in men and women. Despite advances in diagnosing and treating colorectal cancer prognosis remains poor because of persistent mechanisms of tumor proliferation. A new study has zeroed in on one protein that could be behind some of these mechanisms of colorectal cancer spread. JARID1B is a demethylase enzyme encoded by the gene KDM5B and has been implicated in the development of several cancers, including breast, prostate, and liver cancer. Researchers found that JARID1B was significantly upregulated in colorectal cancer tissue versus adjacent normal tissue. In patients with colorectal cancer, high JARID1B expression was associated with poor overall survival. Experiments revealed that JARID1B promoted the spread of colorectal tumor cells through the Wnt/β-catenin signaling pathway. Specifically, by inhibiting the protein CDX2..." The rest of the transcript, along with a link to the research itself, is available on the resource itself.
Material Type: Diagram/Illustration, Reading
This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview: "PD-L1 is a powerful cloaking protein for cancer cells. Abundant PD-L1 on their surface binds with the receptor PD-1 on immune cells. The PD-L1/PD-1 axis transfers inhibitory signaling to the immune system that the cancer cells pose no danger. How this cloaking process unfolds has remained unclear. Now, researchers report the prominence of the enzyme USP22. USP22 is overexpressed in malignant tumors of several types, including those of the lung. Initial experiments on human lung cancer cells showed that USP22 might regulate PD-L1. A closer look revealed that USP22 deubiquitinated and stabilized PD-L1. USP22 enlists the help another protein, CSN5, to stabilize PD-L1. By inhibiting USP22 genetically, researchers could suppress the formation of tumors. Targeting USP22 in the clinic could therefore be one way to decloak cancer cells and make them vulnerable to existing anti-cancer therapies..." The rest of the transcript, along with a link to the research itself, is available on the resource itself.
Material Type: Diagram/Illustration, Reading
This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview: "Farnesyltransferase (FTase) is an enzyme implicated in various diseases, including cancer, hepatitis D, and progeria. One of the two subunits that make up FTase has a proline-rich region, which has been shown to affect signal transduction. Could this region be the key to FTase’s function, especially in disease? To find out, researchers recently explored how modifications altered the behavior of FTase. Phylogenetics revealed that the proline-rich region of FTase is highly conserved in mammals, with the exception of marsupials, which harbor an alanine-rich region instead. Interestingly, when the proline-rich region was removed, FTase retained the ability to bind to its normal physiological partners and did not appear to bind to itself. Indeed, the presence or absence of the proline-rich region did not affect the activity of FTase from either humans or rats. And prominent FTase inhibitors, namely lonafarnib and tipifarnib, worked just as well on either form..." The rest of the transcript, along with a link to the research itself, is available on the resource itself.
Material Type: Diagram/Illustration, Reading
Enzymes are biocatalyst they accelerate the chemical reaction. They are organic, all enzymes are made of protein but not all enzymes are protein. Certain biological reactions can be catalyzed by RNA called Ribozyme. Protein is a dynamic molecule; its activity depends on the three-dimensional structure. For example, the water droplet size gets flexible, if you touch. Protein folding and three-dimensional structures are vital for activity. There is no living cell without an enzyme, in the living cell; it functions to accelerate the biological reaction. There is a misfolded protein infectious agent called Prion, which causes normal brain protein to misfold which, leads to neurodegratative disease. This module presents concise notes of enzyme basic concepts; bioinformatics tools and few examples of enzymes in everyday life.
Material Type: Activity/Lab, Lecture Notes, Module