What is 'innate behaviour'? Where does it feature in the environment? And how does it compare to 'learned behaviour? Learn about it in this video by The Virtual School.
- Material Type:
- Diagram/Illustration
- Lecture
- Date Added:
- 02/22/2013
What is 'innate behaviour'? Where does it feature in the environment? And how does it compare to 'learned behaviour? Learn about it in this video by The Virtual School.
What is 'innate behaviour'? Where does it feature in the environment? And how does it compare to 'learned behaviour? In this Ecology GCSE / K12 video learn all of the answers to these questions.Are you a passionate teacher who would like to reach tens of thousands of learners? Get in touch: vsteam@fusion-universal.comFind out more: http://www.thevirtualschool.comThis video is distributed under a Creative Commons License: Attribution-NonCommercial-NoDerivs CC BY-NC-ND
This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview:
"N6-methyl-adenosine (m⁶A) methylation is the most abundant epigenetic modification on mRNA and lncRNA. This modification is regulated by m⁶A writers, readers, and erasers, and its proper regulation is critical for innate and adaptive immunity, especially since m⁶A can play different roles depending on the transcript region, cell type, or specific regulators involved. For example, in the innate immune system, m⁶A can promote the antitumor and antiviral activity of NK cells. It can also activate disease- fighting M1 macrophages, reduce inflammation, and encourage dendritic cell maturation and activity. On the other hand, m⁶A can prevent dendritic cells from cross-presenting tumor antigens to prime T cells, hindering the antitumor response. In the adaptive immune system, m⁶A promotes T cell homeostasis but also suppresses T follicular helper cell development and it can either exacerbate or inhibit HIV-1 infection in CD4⁺ T cells..."
The rest of the transcript, along with a link to the research itself, is available on the resource itself.